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arterial |
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2002) recently suggested that the mechanism for arsenic-induced cardiovascular disease is the increased susceptibility of platelets to aggregate, resulting in enhanced arterial thrombosis. The third part provides an update on the interventions that have been developed in the catheterization laboratory for the treatment of arterial thrombosis. The company's anti-thrombotic program addresses the unmet need in the prevention of arterial thrombosis (angioplasty, unstable angina, coronary arterial bypass graft, acute myocardial infarction and stroke). |
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