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The organelles such as Golgi complex and endoplasmic reticulum were not observed in the cytoplasm; however, one or two mitochondria were sometimes observed in the cytoplasm (Fig.
Therefore, phagocytic hemocytes often contained abundant numerous mitochondria, Golgi complex, endoplasmic reticulum, and a great quantity of glycogen.
7] Human genes: GALNT3, UDP-N-acetyl-[alpha]-D-galactosamine: polypeptide-N-acetylgalactosaminyltransferase 3; COG7, component of oligomeric golgi complex 7; LMNA, lamin A/C (previous symbols: LMN1, CMD1A); MGAT2, mannosyl (alpha-1,6-)-glycoprotein beta-1,2-N-acetylglucosaminyltransferase; GCS1, glucosidase; SLC35C1, solute carrier family 35, member C1; B4GALT1, UDP-Gal:betaGlcNAc beta 1,4-galactosyltransferase, polypepfide 1; SLC35A1, solute carrier family 35 (CMP-sialic acid transporter), member A1; POMGNTI, protein O-linked mannose beta 1,2-N-acetylglucosaminyltransferase; GNE, glucosamine (UDP-N-acetyl)-2-epimerase/N-acetylmannosamine kinase; GALT, galactose-1-phosphate uridylyltransferase; ALDOB, aldolase B, fructose-bisphosphate.
In the early vitellogenic oocyte, especially with the initiation of yolk formation, lipid droplets were found in the vacuoles formed by the Golgi complex in the perinuclear region (Fig.
In the late vitellogenic oocyte, accumulation of lipid droplets by the Golgi complex, which is composed of the Golgi sac, Golgi vacuole and Golgi vesicle began in the vacuoles and vesicles (Fig.
It was hypothesized that the electron-dense (type 1) granules were phagocytosed foreign materials (microorganisms, or other granules) or internal waste materials (organelles or fragments); the low electron-dense granules (type 2) primary lysosomes, probably originating in the Golgi complex or endoplasmic reticulum; and the medium electron-dense granules, secondary lysosomes, formed by fusion of type 1 and type 2 granules or matured by type 1 granules, which absorbed some hydrolytic enzymes.