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Laboratory experiments have shown that supporting cells from mice can be coaxed into becoming hair cells in part by blocking production of p27.
Distortion-product otoacoustic emissions (DPOAE) are the sounds emitted by the outer hair cells of the cochlea in response to a pair of simultaneously presented pure-tone stimuli.
The damage caused by noise -- over a lifetime or just one evening -- reduces the ability of hair cells to talk to the brain via ribbon synapse connections with nerve cells.
Whereas previous research indicated that hair cells are not replaced, this latest study found that replacement does indeed occur, but at very low levels.
This is an area that's a bit more futuristic and ultimately restoring the hair cells will be the cure.
When it's moving at the same speed as you, the hair cells no longer send a signal to the brain.
The bulk of the damage caused by loud noises is done at the level of the hair cells.
Together with her colleagues, she also unraveled the roles of most of the proteins encoded by these genes, namely in the sensory hair cells, their stimulating gel, and the supporting cells, by multidisciplinary studies of engineered mouse models.
However, the number of inner hair cells which transmit amplified sound signals to the brain, appeared normal.
Humans are born with 30,000 hair cells in each ear.
Free-radical species are produced by NADPH oxidase in the inner hair cells following cisplatin exposure.