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In conclusion, DSF-52 isolated from Artemisia argyi, inhibits the expression of inflammation mediators by targeting NF-[kappa]B, JNK/p38 MAPKs and Jak2/Stat3 pathways.
Aescin also down-regulated levels of inflammation mediators (TNF- [alpha], IL-1 [beta] and NO) and ll [beta]-HSD2 expression in liver, up-regulated GR expression, enhanced endogenous antioxidative capacity, but have no obvious effect on 1l [beta]-HSDl expression in liver.
Sepicalm VG offers a novel mode of action with the dual benefit of the modulation of inflammation mediators and the reduction of the genetic expression of tyrosinase, a key pigmentation enzyme, at the early stage of melano-genesis.