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23) further demonstrated significant urinary calcium, urinary sulfate, and net acid excretions in an animal protein-rich diet relative to ovo-vegetarian and vegetarian diets.
Our most advanced product candidate for the treatment of hyperuricemia and gout, lesinurad is an oral, once-daily inhibitor of URAT1, a transporter in the kidney that regulates uric acid excretion from the body.
and uric acid excretions, glomerular and proximal tubular function, as well as [Na.
Our most advanced product candidate, RDEA594, is a selective inhibitor of URAT1, a transporter in the kidney that regulates uric acid excretion from the body.
Our most advanced product candidate for the treatment of hyperuricemia and gout, RDEA594, is an oral, once-daily inhibitor of URAT1, a transporter in the kidney that regulates uric acid excretion from the body.
Circadian periodicity of urinary volume, creatinine and 5-hydroxyindole acetic acid excretion in healthy Indians.
Our most advanced product candidate, RDEA594, is an inhibitor of URAT1, a transporter in the kidney that regulates uric acid excretion from the body.
RDEA594 lowers excessive uric acid levels (the underlying cause of gout) by increasing uric acid excretion, while allopurinol decreases its production.
Abnormal formiminoglutamic acid excretion was related not only to placental abruption and spontaneous abortion but also to adverse outcomes in previous pregnancies, including prematurity, congenital defects, and perinatal mortality.
RDEA594 is an inhibitor of URAT1, a transporter in the kidney that regulates uric acid excretion.
RDEA594 exhibits a concentration-dependent inhibitory effect on the URAT1-mediated uptake of uric acid ex vivo and increases uric acid excretion in animal models.
Characteristically, a 20- to 200-fold increase in N-acetylneuraminic acid excretion in urine has been described in infantile cases of the disease.