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Hence, ACC represents a triple threat to placental perfusion and foetal well-being: brachial artery pressure may decrease due to decreased cardiac output, uterine artery pressure may decrease below brachial artery pressure and uterine venous pressure may increase.
The decreased cardiac output stimulates sympathetic activity and blood volume expansion via the activation of the reninangiotensin-aldosterone axis.
A patient who is hypovolemic, or a patient who has decreased cardiac output from an MI, will exhibit a decrease in the systolic pressure.
However, in fulminant cases, impaired venous return and decreased cardiac output might result in potentially fatal hypotension.