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Related to envenoming: envenomation, Antivenom, envenomate
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granulatus envenoming is associated with a higher morbidity and mortality compared with P.
The spectrum and degree of symptoms and signs of systemic envenoming are determined by several factors:
Organophosphate as well ascarbamate poisoning and scorpion envenoming can cause a state of anxiety and restlessness, muscle twitching, increased oropharyngeal secretions, decrease in motor power, raised blood pressure, tachycardia and respiratory distress.
In most cases the degree of severity of scorpion envenoming is evident quite soon after the sting (within 15 - 60 minutes).
Despite the lack of published data, mortality and morbidity after Tiger snake envenoming has fallen with appropriate first-aid pressure immobilisation, monitoring of the patient and biochemical markers and the use of antivenom.
About 50% of Tiger snake bites result in significant envenoming and prior to antivenom therapy there was a mortality of 45% (1).
The aim of this study was to describe the severity, clinical course, complications and management in a group of patients with definite Tiger snake envenoming with coagulopathy.
there was a positive Commonwealth Serum Laboratories Venom Detection kit (VDk) result to Tiger snake or if not, reversal of envenoming with Tiger snake antivenom.
The aggressive and progressive cytotoxic nature of envenoming is usually evident within hours of the bite.
Abnormal blood biochemistry, such as raised serum concentrations of creatine kinase and other muscle-derived enzymes, is commonly found in severe envenoming because of local muscle damage.
Snake venom ophthalmia is eye envenoming that occurs when venom is spat into the eyes (see under ancillary treatment).
In neurotoxic/cytotoxic berg adder bite the cytotoxic component of envenoming is quite prominent when compared with the minimal local effects of elapid neurotoxic snake bite.