References in periodicals archive ?
Antigen-induced, tolerogenic CD11c+, CD11b+ dendritic cells are abundant in Peyer's patches during the induction of oral tolerance to type II collagen and suppress experimental collagen-induced arthritis.
RELATED ARTICLE: HOW UC-II[R] INDUCES ORAL TOLERANCE
Oral tolerance and its relation to food hypersensitivities.
Under the oral tolerance scenario, almost any food antigen could be a candidate for therapy.
Optiquel[TM] therapy is based on oral tolerance, an immunologic mechanism that down-regulates immune responses in an antigen-specific manner, rather than a non-specific immunosuppressive approach.
But Weiner hasn't given up on the concept of oral tolerance.
Although reminiscent of 19th-century snake oil medicine, oral tolerance therapy--persuading a person's body to accept foreign proteins by feeding those proteins to the person--may offer a specific, extremely safe way to treat a host of autoimmune diseases.
arGentis researchers believe that the SNP maker may be applicable to achieving oral immune tolerance in many, if not all, autoimmune diseases, providing a platform for developing oral tolerance therapeutics for large subpopulations of those diseases.
The interaction between gut-associated lymphoid tissue in the duodenum and epitopes of orally administered undenatured type II collagen facilitates oral tolerance to the antigen and stems systemic T-cell attack on joint cartilage.
The defense against hypersensitivities to dietary proteins is a well-developed, multitier system dependent on an effective mucosal barrier in association with the oral tolerance generated by the cellular immune system of the GALT.