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In contrast, after exposure to 2500 (sd 500) lx light (420-800 nm) for 12 h, RPE cells in the positive control (light exposure, no BAE treatment) exhibited premature senescence, low (P < 0*01) cell viability and increased (P < 0*01) vascular endothelial growth factor (VEGF) release compared with negative control cells, which were not subjected to light irradiation and BAE exposure.
By promoting premature senescence in the normal HMEC, the radiation treatments accelerated the outgrowth of the vHMECs.

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