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Mutations in subunits of the epithelial sodium channel cause salt wasting with hyperkalaemic acidosis, pseudohypoaldosteronism type 1.
Endocrine and metabolic syndromes in the critically ill: Cerebral salt wasting.
Cerebral salt wasting shows an excessively high urinary sodium concentration, >100 mmol/L (4).
PHA-I is a rare genetic disease where inactivating mutations of NR3C2 gene coding for mineralocorticoid receptor may lead to a dysfunction of ion channels resulting in salt wasting crisis and life threatening hyperkalaemia in the early neonatal period.
Inadequate mineralocorticoid replace ment leads to renal salt wasting, hyperkalemia, and hyponatremic dehydration.
The opposite is usually true for a contracted extracellular fluid volume (such as occurs with diuretics, primary adrenal insufficiency, and salt-wasting nephropathy), although a caveat is that cerebral and some forms of renal salt wasting can also cause renal uric acid loss.