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EPILEPSY, med. jur. A disease of the brain, which occurs in paroxysms, with uncertain intervals between them.
     2. These paroxysms are characterized by the loss of sensation, and convulsive motions of the muscles. When long continued and violent, this disease is very apt to end in dementia. (q.v.) It gradually destroys the memory, and impairs the intellect, and is one of the causes of an unsound mind. 8 Ves. 87. Vide Dig. 50, 16, 123; Id. 21, 1, 4, 5.

References in periodicals archive ?
The Neural Correlates of Childhood Absence Epilepsy
The findings showed that when compared with patients who had been diagnosed with childhood or juvenile absence epilepsy (AE), those with JME in their mid-40s were as likely or more likely to have a college degree, steady employment, and full integration into their social context.
It presents similarly to childhood absence epilepsy, but after age 10 years.
Effects of GABAB receptor antagonists on learning and memory retention in a rat model of absence epilepsy.
Used for infantile spasms and Landau-Kleffer syndrome, Lennox-Gastault syndrome, absence epilepsy, progressive myoclonic epilepsy, Rasmussen's syndrome, epilepsia partialis continua
Paroxysmal tonic upgaze of childhood and childhood absence epilepsy.
18, 20) En el caso de la rata, el sujeto de interes en esta revision, existen dos cepas de ratas que presentan espontaneamente descargas espiga-onda similares a las observadas en las crisis de ausencia: las Ratas Geneticamente Propensas a Crisis de Ausencia de Estrasburgo (GAERS, siglas para Genetic Absence Epilepsy Rat from Strasbourg) y las Ratas Wistar Albino Glaxo (WAG/Rij, siglas para Wistar Albino Glaxo Rat from Rijswijk).
A relatively less-common condition called childhood absence epilepsy (CAE) may easily be misdiagnosed as ADHD, especially the predominantly inattentive type.
The rarity of adult onset absence epilepsy over the age of 25 years suggested that either this syndrome was exceedingly rare or under-recognized in all populations including ours.
Their topics include the role of the somatosensory cortex in pain processing, rehabilitation after stroke using brain-computer interfaces and neurostimulation, mechanism of epileptogenesis in the somatosensory cortex in rats with genetic absence epilepsy, thermo-nociceptive responses evoked by laser pulses in the primary somatosensory cortex, and the regulation of the critical period for whisker lesion-induced barrel structural plasticity in the mouse somatosensory cortex.