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Consistent with our result, one cleavage fragment within the PRR, derived from the shorter variant form (isoform 2) of human ITIH4, was also found to be decreased in pancreatic cancer (19).
The N- and C-terminal sets of peptides were individually normalized, providing a semiquantitative measure of the abundance of N- and C-terminal cleavage fragments of NT-proBNP in the circulation of HF patients.
This can occur when small molecules act as haptens or if exposure can cause novel cleavage fragments to which the immune system was ignorant.