The protective effect of granulocyte colony-stimulating factor on endometrium and ovary in a rat model of diabetes mellitus.
Granulocyte colony-stimulating factor with or without stem cell factor extends time to premature ovarian insufficiency in female mice treated with alkylating chemotherapy.
Baby developed severe neutropenia at DOL 2, with absolute polymorphonuclear count <500 [micro]/L, and was started with granulocyte colony-stimulating factor. Due to favorable clinical evolution in addition to negative blood culture, Ampicillin-Gentamicin were discontinued at DOL 5.
Early treatment of neutropenia with granulocyte colony-stimulating factor might play an important role in the rapid increase of neutrophil count with no changes in short term mortality.
It is assumed that about 20% of cases are related to autoimmune disorders and about 40-50% of cases are unexplained RM G-CSF (Granulocyte Colony-stimulating Factor) is a glycoprotein that is secreted from endothelial cells, macrophages and some other immune system cells (6).
Granulocyte-macrophage colony-stimulating factor (GM-CSF) and granulocyte colony-stimulating factor (G-CSF) secretion by adherent monocytes measured by quantitative immunoassays.
Granulocyte
colony-stimulating factor (G-CSF) is a glycoprotein that stimulates the bone marrow to produce granulocytes and stem cells and release them into the blood.
Derman, "A randomized trial of granulocyte-macrophage
colony-stimulating factor in neonates with sepsis and neutropenia," Pediatrics, vol.
In recent years some studies have stated that intrauterine infusion of granulocyte
colony-stimulating factor (GCSF) may be effective in patients with treatment-resistant thin endometrium.
Granulocyte
colony-stimulating factor (G-CSF) is a glycoprotein first recognized for its ability to facilitate the formation of neutrophilic granulocyte colonies in soft agar from bone marrow cells [1, 2].
Colony-stimulating factors (CSFs): Cytokines that stimulate the production of blood cells.
Hamilton, "The
colony-stimulating factors and collagen-induced arthritis: exacerbation of disease by M-CSF and G-CSF and requirement for endogenous M-CSF," Journal of Leukocyte Biology, vol.