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According to these cases, they described the course of the facial corticobulbar tract as consisting of looping fibers that descend at least to the medullary level and then decussate.
PBA is most common in diseases that interfere bilaterally with the corticohypo-thalamic and corticobulbar tracts that control voluntary and involuntary faciorespiratory mechanisms.
This results in compression of the crus and subsequent damaging of the descending corticospinal tracts and corticobulbar tracts (Kernohan & Woltman).