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An event, circumstance, influence, or element that plays a part in bringing about a result.

A factor in a case contributes to its causation or outcome. In the area of Negligence law, the factors, or chain of causation, are important in determining whether liability ensues from a particular action done by the defendant.

West's Encyclopedia of American Law, edition 2. Copyright 2008 The Gale Group, Inc. All rights reserved.


n. 1) a salesman who sells in his/her own name on behalf of others, taking a commission for services. 2) something that contributes to the result.

Copyright © 1981-2005 by Gerald N. Hill and Kathleen T. Hill. All Right reserved.


1 a mercantile agent. An agent who is in the ordinary course of business entrusted with goods or documents of title representing goods with a view to their sale. A factor has a lien over goods entrusted to him; this lien covers any claims he may have against his principal arising out of the agency. Most factors will be mercantile agents (and have the powers of such) for the purposes of the Factors Act 1889. Under this Act, in certain circumstances a factor may pass a good title to goods entrusted to him.
2 an institution to whom a company assigns its book debts (see FACTORING).
3 in Scotland a landlord or superior's agent.
Collins Dictionary of Law © W.J. Stewart, 2006
References in periodicals archive ?
GM-CSF regulates alveolar macrophage differentiation and innate immunity in the lung through PU.1.
Second, statistical differences (P<0.05) were found in the serum levels of cytokines GM-CSF, IFN-[gamma], and KC (HFD vs HFD-FR).
However, the results of studies are not consistent and a study in mice by Kong et al22 did not reveal any positive effect of G-CSF or GM-CSF on atherosclerosis, instead it was shown by the study that G-CSF and GM-CSF induce atherosclerosis in mice.
They found that the mice that had been given large amounts of a special cytokine - molecules that warn other cells that there's an infection or other trauma in the body - called GM-CSF, had better survival and lung function than the other mice.
In this study, we optimized and compared the yield and purity of murine derived CD11c+ BMDCs cultured by flask culture method and CD11c Positive Selection kit based method, in the presence of low dose of GM-CSF in nontreated tissue culture flasks.
Herein, we assessed cytokines and growth factors crucial for eosinophil proliferation and differentiation (GM-CSF, IL5, and IL4), for their release from bone marrow (IL5), for their survival (IL5, IL13, GM-CSF, and IL4) and priming in circulation (IL5, GM-CSF, IL4, and TNF[alpha]), and for extravasation (IL4, IFN[gamma], TNF[alpha], IL8, and eotaxin) and homing into the gut (eotaxin, RANTES, IL5, IL8, IL13, GM-CSF, and TNF[alpha]) as well as responsible for inducing production and secretion of their mediators (TNF[alpha], IFN[gamma], IL-5, GM-CSF, and eotaxin) (reviewed in [10, 17, 20]).
Blood sample was tested for antibodies against granulocyte macrophage-colony stimulating factor (GM-CSF) through an outside lab and was reported negative.
Granulocyte macrophage colony stimulating factor (GM-CSF) also contributes to the enhancement of COPD through increasing the number of neutrophils [26, 27].
In addition, EPC is regulated by cytokine such as NO, VEGF, GM-CSF, IL-8, and MCP-1 as well [15-20].
In RA, cytokines such as tumor necrosis factor alpha (TNF-[alpha]), interleukin-6 (IL-6), IL-17a, IL-22, IL-23, IL-1[beta], IL-8, interferon-y (IFN-[gamma]), granulocyte/macrophage colony-stimulating factor (GM-CSF), granulocyte colony stimulating factor (G-CSF), IL-15, IL-18, IL-33, and IL-37 [11, 13, 18-36] are all detected either in the serum or in the synovial fluids of these patients.