Table-2: Analysis of the risk factors associated with Graves' disease
Predictors of time to remission and treatment failure in patients with Graves' disease
treated with propylthiouracil.
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Thyroid stimulating auto-antibodies (TSAb)--that is, TSI--are the direct cause of Graves' disease
, while thyroid blocking antibodies (TBAb), which inhibit TSH binding to the thyroid receptor, can cause hypothyroidism.
Increased aggressiveness of thyroid cancer in patients with Graves' disease
According to the authors, vitamin D deficiency could exacerbate the development of Graves' disease
Fetal hyperthyroidism: Thy roid-stimulating hormone receptor-stimulating antibodies, which occur and reach high levels in mothers who exhibit a picture of Graves' disease
, cross the placenta and lead to a picture of fetal hyperthyroidism because they have a structure like immunoglobulin-G.
Comparison of methimazole and propylthiouracil in patients with hyperthyroidism caused by Graves' disease
Induction of murine neonatal tolerance against Graves' disease
using recombinant adenovirus expressing the TSH receptor A-subunit.
also seemed not to affect hand grip strength, dexterity, and function compared with patients with primary hyperthyroidism but without Graves' disease
Should Graves' disease
be considered a collagen disorder of thyroid, skeletal muscle and connective tissue?
5) Concurrent carcinoma was more frequent in patients with toxic adenoma (8 %) than in those with Graves' disease
(6 %) and toxic nodular goiter (5 %).