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An increase in the incidence of Jarisch-Herxheimer reactions has been described among patients with malignant syphilis and patients coinfected with HIV in general.
Isolated ocular Jarisch-Herxheimer reaction after initiating tuberculostatic therapy in a child.
* A Jarisch-Herxheimer reaction is characterized by hypotension, tachycardia, chills, rigors, diaphoresis, and elevated body temperature and can occur after initial antibiotic therapy for infections caused by spirochetes, including relapsing fever (1).
Another explanation may be that lysis of trypanosomes triggered an immunological response, comparable with the Jarisch-Herxheimer reaction [14].
The Jarisch-Herxheimer reaction is a dramatic reaction to treatment, occurring within 2 hours of the dose, caused by the killing of syphilitic spirochetes in the blood and the release of endotoxins into the tissues, she said.
The Jarisch-Herxheimer reaction is marked by fever, chills, headache, and myalgia occurring 2-24 hours following penicillin therapy.
Within hours of the initiation of antibiotic, he developed a Jarisch-Herxheimer reaction with fevers, chills, and tachycardia which resolved within one day.
During the first 24 hours after initial treatment, patients may develop an acute febrile illness known as the Jarisch-Herxheimer reaction. This is largely the result of massive lysis of the pathogen, spilling large quantities of inflammatory cytokines into the bloodstream.
Because of thrombocytopenia, a peripheral blood smear was examined, revealing spirochetes diagnostic of TBRE Her transient hypotension was attributed to a Jarisch-Herxheimer reaction (JHR).