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CELL. A small room in a prison. See Dungeon.

References in periodicals archive ?
PRLR and 3[beta]-HSD were observed in both small and large luteal cells. A qualitative estimation indicated that the maximum colocalization of PRLR and 3[beta]-HSD occurred during midpregnancy, and this result is consistent with what was analyzed for each marker separately both by immunohistochemistry and by qPCR.
The antioxidant beta-carotene prevents covalent cross-linking between cholesterol side-chain cleavage cytochrome P450 and its electron donor, adrenodoxin, in bovine luteal cells. Mol.
Stevenson, "Changes in enzyme activities during the artificially stimulated transition from follicular to luteal cell types in rat ovary," European Journal of Biochemistry, vol.
Mukhopadhyay, "Lysophosphatidic acid antagonizes the morphoregulatory effects of the luteinizing hormone on luteal cells: possible role of small Rho-G-proteins, " Biology of Reproduction, vol.
Oxidative stress--inducible antioxidant adaptive response during prostaglandin F2 alpha--induced luteal cell death in vivo.
Carroll DJ, Grummer RR, Mao FC (1992) Progesterone production by culture luteal cells in the presence of bovine low and high-density lipoproteins purified by heparin affinity chromatography.
Responsiveness of porcine large and small luteal cells to luteotropic or luteolytic hormones and cell morphology changes during the estrous cycle and pregnancy.
After ovulation takes place, for a couple of days the former follicle--now the corpus luteum--fills up with these luteal cells, which make progesterone.
Kaminski, "The influence of adiponectin on the transcriptomic profile of porcine luteal cells," Functional & Integrative Genomics, vol.
It is also suggested that the increased PI between 48 and 72 h during GCs real-time culturing may be a result of granulosa cell differentiation into luteal cells, which takes place both in vivo and in vitro.
In addition, galectin1 and -3 may mediate progesterone production and metabolism in luteal cells via different mechanisms (Nio-Kobayashi & Iwanaga, 2010).
Immunohistochemistry depicted the presence of the ligands LEP and GHR, together with their respective receptors, LEPR and Ghr-R1A, in equine luteal cells. No staining was present in negative controls (Figures 1(e) and 1(f)).