(redirected from Nerve terminal)
Also found in: Dictionary, Thesaurus, Medical, Encyclopedia.
Related to Nerve terminal: synaptic cleft, Synaptic knob
References in periodicals archive ?
Second, asynchronous conduction increases due to an immature motor nerve terminal and neuromuscular junction following denervation-reinnervation; thus, the duration of excitatory signal propagation from muscle fibers to the recording needle significantly varied, allowing more waveform spikes to be obtained.
Suzuki et al (2) suggested that a gradual decrease in the amount of transmitter released from the presynaptic nerve terminals during TOF stimulation was the explanation for the delayed recovery of the TOF ratio, despite no delay in T1 recovery, although in their study, the duration of the operation and the supplemental doses of vecuronium and mepivacaine given were not detailed (2).
Pudendal nerve terminal motor latency (PNTML) was also assessed.
Pestronk, "Mechanisms of postsynaptic plasticity: remodeling of the junctional acetylcholine receptor cluster induced by motor nerve terminal outgrowth," Journal of Neuroscience, vol.
A third pattern, damage to neuritic projections, produces a decrement in the membrane/total protein ratio in the nerve terminal region but an increase in areas where reactive sprouting takes place (Kostrzewa and Jacobowitz 1974; Navarro et al.
For example, kinesin ferries neurotransmitters down the extensions of nerve cells, whereas dynein, another motor protein, shuttles cargo in the opposite direction, from the nerve terminal bac| to the center of the cell, says Ronald D Vale, a cell biologist at the University of California, San Francisco.
No changes in anal manometry, sphincter integrity upon endoanal ultrasound, or pudendal nerve terminal motor length were noted.
Because HC-3 binding is responsive to cholinergic nerve impulse activity, whereas CHAT is a static marker for nerve terminals, the ratio of HC-3 binding to ChAT activity represents an index of activity per nerve terminal (Aubert et al.
Neuropathic pain responds to drugs that produce a blockade of neuronal sodium channels, inhibiting nerve terminal release of dopamine, [gamma]-aminobutyric acid, and glutamate.
Patients in this series were identified using a complete urologic evaluation, sigmoidoscopy, anal manometry, anal sonography, and pudendal nerve terminal motor latency studies.
Choline acetyltransferase (ChAT) was used as a marker for nerve terminal cytoplasm and was measured by means of a radiometric assay described by Fonnum (1975), with the following modifications.