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Association between the DQA MHC class II gene and Puumala virus infection in Myodes glareolus, the bank vole.
Behavioral, physiologic, and habitat influences on the dynamics of Puumala virus infection in bank voles (Clethrionomys glareolus).
Hantaan virus and Dobrava virus typically produce severe disease, whereas Seoul virus produces moderate disease and Puumala virus produces mild disease.
The humoral response to puumala virus infection (nephropathia epidemica) investigated by viral protein specific immunoassays.
at NRC ([double dagger]) IgM PUUV 1 - 2 - ND 3 - ND * E, equivocal; IFA, indirect fluorescent antibody; ND, not done; NRC, National Reference Center for Hantavirus (Lyon, France); PUUV, Puumala virus, SEOV, Seoul virus; +, positive; -, negative.
Puumala virus (PUUV) causes most hantavirus disease cases in Central and Western Europe and is the only human pathogenic hantavirus in Fennoscandia (1).
Puumala virus (PUUV) (family Bunyaviridae) is an enveloped hantavirus that contains a single-stranded trisegmented RNA genome of negative polarity (1).
For patients presumed to have CNS symptoms, samples were screened for antibodies against a panel of meningoencephalitis agents (HSV, varicella zoster virus, human herpesvirus 6, enterovirus, and Mycoplasma pneumoniae bacteria); for patients presumed to have febrile illness, samples were screened for Puumala virus.
All serum samples exhibited substantially higher neutralizing titers toward DOBV than toward Puumala virus, Hantaan virus, and Seoul virus.
Puumala virus (PUUV), by far the most frequent cause of hantavirus disease in Germany (7), causes a milder form of HFRS (8) called nephropathia epidemica (NE).