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AIR. That fluid transparent substance which surrounds our globe.
     2. No property can be had in the air it belongs equally to all men, being indispensable to their existence. To poison or materially to change the air, to the annoyance of the public, is a nuisance. Cro. Cr. 610; 2 Ld. Raym 1163; I Burr. 333; 1 Str. 686 Hawk. B. 1, c. 75, s. 10; Dane's Ab. Index h.t. But this must be understood with this qualification, that no one has a right to use the air over another man's land, in such a manner as to be injurious to him. See 4 Campb. 219; Bowy. Mod. Civ. Law, 62; 4 Bouv. Inst. n. 36 1; Grot. Droit de la Guerre et de la Paix, liv. 2, c. 2, Sec. 3, note, 3 et 4.
     3. It is the right of the proprietor of an estate to enjoy the light and air that will come to him, and, in general, no one has a right to deprive him of them; but sometimes in building, a man opens windows over his neighbor's ground, and the latter, desirous of building on his own ground, necessarily stops the windows already built, and deprives the first builder of light and air; this he has the right to do, unless the windows are ancient lights, (q.v.) or the proprietor has acquired a right by grant or prescription to have such windows open. See Crabb on R. P. Sec. 444 to 479 and Plan. Vide Nuisance.

References in periodicals archive ?
The fact that diphenhydramine, a histamine antagonist, reduced airway symptoms indicates that brevetoxins activate histamine-producing cells, such as mast cells and basophils.
The results of his second sleep study were worse than the first, but he ultimately obtained excellent relief with bilevel positive airway pressure.
When the team looked at a model of an asthmatic lung, with its thickened and inflamed airway walls, a dramatic increase in airflow resistance occurred with the same constriction of smooth muscles, he says.
Increased airway kinin levels occur after exposure to a variety of noxious stimuli (Lauredo et al.
The purpose of this article is to report our own findings and to review the clinical presentation of acute supraglottitis, microbiology, airway management, and treatment.
Airway hyperractivity is not asthma, but it may lead to an understanding of how airway hyperractivity develops into asthma.
Many researchers have proposed hypotheses for the cause of the airway obstruction in Pierre Robin sequence.
There are a number of ways to evaluate the upper airway (table 1).
It also involves state-specific changes in respiratory load compensation in the form of variations in the medullary motor output to primary and accessory respiratory muscles in response to upper airway resistance and changes in resistance.
This increased responsiveness can take the form of swelling of the airway wall, excess mucus production that can clog the airways, and significant narrowing of the airways when tiny muscles in the airway wall, called "airway smooth muscle", contract.