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Related to aplastic crisis: aplastic anemia, sequestration crisis
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Patients with HS are at an increased risk of developing perioperative complications, such as aplastic crisis and hemolytic episodes, for which awareness and vigilance are important.
Acute splenic sequestration together with aplastic crisis caused by human parvovirus B19 in patients with sickle cell disease.
HPVB19 infection rarely presents any symptoms in most immunocompetent individuals, but it causes several well-known clinical manifestations, including erythema infectiosum, arthropathy, transient aplastic crisis, nonimmune hydrops fetalis, meningitis, encephalitis, and myocarditis, particularly in childhood [10].
Knowing HPV B19 as the most common causative agent in the development of aplastic crisis in hemolytic anemias; specifically hereditary spherocytosis polymerase chain reaction (PCR) for HPV B19, DNA was performed which was positive in all three patients (Figures 1 and 2).
Hematologic diseases such as transient PRC aplastic crisis, hydrops fetalis and single or multiple transient parvovirus-related cytopenias (neutropenia, autoimmune thrombocytopenic purpura, pancytopenia) are also seen in immunocompetent patients (4), (5), (7-10), (39).
An aplastic crisis can occur when the bone marrow and cell production fail.
Infection with parvovirus B19, although frequently asymptomatic, may result in erythema infectiosum, arthropathy, pregnancy complications (e.g., hydrops fetalis), transient aplastic crisis, and disease in immunocompromised patients (2).
* Aplastic crisis. In an aplastic crisis, parvovirus p19 causes severe anemia by attacking and destroying red blood cell progenitors.