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Factor VII is the first coagulation protein to decrease when there is a hepatocyte damage, probably because of its short half-life (2-4h) [28,29].
The disease is caused by deficient or defective blood coagulation proteins, known as factor VIII or IX.
Thrombin, factors Xa and VIIa, in addition to their roles in activating coagulation protein zymogens, can interact with specific cell receptors and activate intracellular signaling pathways that mediate inflammatory responses.
Recombinant factor VIII and factor IX products, designed for hemophilia patients with deficiencies of these respective circulating coagulation proteins, are not derived from human plasma.