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Related to duplications: Genome duplication
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More than half of all colonic duplications are cystic and have very different attributes from the tubular colorectal duplications, as they do not communicate with the adjacent lumen.
Bladder duplication is a very rare congenital malformation of the urinary system, which is classified as complete or incomplete.
Gastric foregut duplication cyst is a rare congenital disease.
Most of the adult intestinal duplications are asymptomatic and remain undiagnosed for years.
Following fish specific genome duplication (FSGD), gene duplicates undergo divergent pathways in gene evolution.
4) The majority of duplications are cystic in nature (53-85%), and the remaining are tubular and generally larger in size, with a tendency to communicate with the contiguous gut lumen.
sup][2] The etiology of intestinal duplications is unknown, although more than 80% occur in children aged < 2 years.
Foregut duplications are cystic lesions that develop from the primitive esophagus during embryogenesis between the fourth and eighth week of gestation.
Duplications and deletions, collectively known as copy number variants, have shaped more than 7 percent of the human genome.
4 According to Effman classification urethral duplications are divided into three main groups as Type I II and III (Figure-2).
Duplications of the alimentary tract are congenital structures that may arise anywhere from the mouth to the anus.
Gastric duplications, the least common among all duplications, constitute 2-7% of GI duplications and mostly present with GI obstruction symptoms, ulceration, and painless hemorrhage, mostly in early ages.