Cell

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CELL. A small room in a prison. See Dungeon.

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TGF-[sz] is the strongest profibrotic factor in EMT of peritoneal mesothelial cells (PMCs) and a key mediator in dialysis-related peritoneal fibrosis.[sup][5],[22] Platelet-derived growth factor (PDGF) also induced partial EMT in vivo and stimulated human PMC proliferation in vitro .[sup][23],[24]As TGF-[sz]R and PDGF receptors (PDGFR) are both glycosylated, and CF is their common posttranslational modification,[sup][25] we believed that blocking CF may simultaneously inhibit TGF-[sz] and PDGF signaling pathways, resulting in a synergistic protective effect in ameliorating EMT.
Yue et al., "Effect of ligustrazine nanoparticles nano spray on transforming growth factor-[beta]/Smad signal pathway of rat peritoneal mesothelial cells induced by tumor necrosis factor-[alpha]," Chinese Journal of Integrative Medicine, vol.
LA exposure also induces production of autoantibodies to mesothelial cells (Marchand et al.
Topley, "Superinduction of IL-6 synthesis in human peritoneal mesothelial cells is related to the induction and stabilization of IL-6 mRNA," Kidney International, vol.
Alternatively, since both amphibole and chrysotile asbestos can cause oxidative stress and cell death in macrophages and mesothelial cells [91, 92], the mechanism of cell activation and apoptosis may be different [72], leading to different pathways of protein degradation.
Asbestos is thought to induce mutations in mesothelial cells by damaging the mitotic spindle (6,7).
In vitro studies with fibroblasts and with human peritoneal mesothelial cells (HPMC) have shown that GDPs impair cellular growth, reduce cell viability, and significantly decrease cellular release of cytokines or chemokines into the incubation medium (Mortier, De Vriese, Schaub, Passlick-Deetjen, & Lameire, 2001; Struik & Douma, 1998; Topley, 1998; Wieslander, Nordin, Kjellstrand, & Boberg, 1991; Witowski et al., 2001).
It should also be emphasized that the multiplicity of proangiogenic agents present in ascites as well as those hypersecreted by stimulated endothelial cells provides evidence that the proangiogenic microenvironment within the peritoneal cavity, including milky spots with a high density of blood vessels [23], is created not only by resident mesothelial cells [24].
Still the cyst was classified as a false cyst based on the lack of an endothelial or mesothelial cell lining.
Isolation of pleural mesothelial cells (PMCs) was performed as described previously.
Examination of effusion cytology is tricky as morphology of reactive mesothelial cells and malignant cells closely resembles.
The expression of both epithelial and mesenchymal markers is a constant feature in malignant mesothelioma, which may be related to the origin of mesothelial cells.[sup.8] The histopathologic differential diagnoses of malignant urogenital mesotheliomas includes metastatic adenocarcinoma, papillary mesothelial hyperplasia, carcinoma of the rete testis and embryonal carcinoma.