excitability

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The result for both is detrusor muscle excitability, causing OAB symptoms (Madhuvrata, Cody, Ellis, Herbison, & Hay-Smith, 2012).
This could be attributed to the common factors affecting type I and type II fibers, such as, reduced ATP production due to the reduced substrate availability, accumulation of metabolic end products that impair contractile events, changes in intracellular and extracellular muscle electrolyte concentration that reduce muscle excitability, and alterations in sarcoplasmic reticulum calcium handling properties23.
Local anaesthetics interfere indirectly with neuromuscular transmission by inhibiting acetylcholine release or by changing the acetylcholine receptor to depress muscle excitability (1,9-12).