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Related to myeloid cell: lymphoid cell

CELL. A small room in a prison. See Dungeon.

A Law Dictionary, Adapted to the Constitution and Laws of the United States. By John Bouvier. Published 1856.
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Cancer recruit harmful myeloid cells, while suppressing helpful ones
We observed that the depletion of [Gr-1.sup.+] myeloid cells prevented the resolution of bleomycin-induced pulmonary fibrosis, whereas their mobilization from the bone marrow or the adoptive transfer of isolated [Gr-1.sup.+] myeloid cells either prior to or after bleomycin challenge enhanced the rate of fibrotic resolution in this model via the TRAIL-dependent apoptosis of myofibroblasts.
Recently, neutrophils have emerged as new tumor-infiltrating myeloid cells, playing an important role in tumor growth and progression [96].
Because myeloid cells copiously express MPO and because halogenated DNA may induce both genetic and epigenetic changes that contribute to carcinogenesis, halogenative stress may account for benzene-induced bone marrow disorders and myeloid leukemia.
The peripheral blood smear presents with a leukoerythroblastic picture, which includes teardrop RBCs, poikilocytosis, anisocytosis, nucleated red blood cells, and immature myeloid cells. Approximately 50% of patients have been found to have the JAK2 V617F mutation.
In contrast, HC is a glycosylated serum protein of ~68 kDa that is produced mainly by myeloid cells, binds ~80%-94% of the endogenous plasma vitamin [B.sub.12], and is largely saturated with vitamin [B.sub.12] (1, 2).
Myeloid cells are regarded to be mainly detrimental in autoimmune diseases of the CNS as they promote neuroinflammation, demyelination, and neurodegeneration [8].
The myeloclysplastic syndromes (MDS) are clonal diseases of hematopoietic stem cells which result in cytopenia(s) and abnormal cell production in the myeloid cell lines.
(2.) Byrd JC, Edenfield WJ, Dow NS, Aylesworth C, Dawson N: Extramedullary myeloid cell tumors in myelodysplastic-syndromes: Not a true indication of impending acute myeloid leukemia.
Because targets of CNIs are NFAT and MAPK pathways, which are widely used signaling by myeloid cells, it is not surprising that CNIs affect myeloid cell functions including MDSCs.
[CD11b.sup.+] myeloid cell populations included [Ly6G.sup.high][Ly6C.sup.low] and [Ly6G.sup.low][Ly6C.sup.high] subpopulations by day 7 after CLP (Figure 1(a)).
Gery et al., "BCOR regulates myeloid cell proliferation and differentiation," Leukemia, vol.