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The electrocardiogram (ECG) shows ST segment elevation or T wave inversions across the anterior precordial leads.
Brugada syndrome is a well-known inherited syndrome characterized by Brugada ECG patterns in precordial leads V1 to V3 that can predispose individuals to ventricular arrhythmias and sudden cardiac death.
Brugada syndrome was first described in 1992 and is characterised by specific electrocardiogram (ECG) changes in the right precordial leads, a structurally normal heart and susceptibility to ventricular arrhythmias (1).
The ECG typically shows a RBBB block pattern with an inferior axis and a QRS transition in precordial leads V3 and V4.
The following case report illustrates some of the problems associated with the diagnosis of BRS in a patient who presents with syncope and ST-segment elevation (ST-SE) in the right precordial leads ([V.
007) enhanced occlusion-induced peak ST-segment elevation in precordial leads [V.
According to the WHO criteria and the Consensus Document of the Joint European Society of Cardiology /American College of Cardiology Committee for the Redefinition of Myocardial Infarction (15), we defined AMI as a typical increase and gradual decrease (cardiac troponin) or more rapid increase and decrease (CK-MB) of biochemical markers of myocardial necrosis with at least one of the following: typical chest pain, ST-segment elevations of at least 2 mm in two or more precordial leads or of at least 1 mm in two or more corresponding extremity leads, and development of pathologic Q-waves on the ECG.
Brugada syndrome is characterized by right bundle branch block and unusual ST-segment elevation in the right precordial leads.
The ECG changes associated with PE include sinus tachycardia (the most common abnormality), complete or incomplete right bundle branch block, a right ventricular strain pattern (T wave inversions in the right precordial leads [V1-4] [+ or -] the inferior leads [II, III, aVF]), a right axis deviation, or an SI QIII TIII pattern (a deep S wave in lead I, Q wave in III, and inverted T wave in III) (2).
The ST segment elevation in precordial leads at presentation was sustained and on follow up patient had sustained ventricular tachycardia attacks which were successfully treated with electrical cardioversion (Fig.