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Mamu-A*01 allele-mediated attenuation of disease progression in simian-human immunodeficiency virus infection.
In a Center for AIDS Research (CFAR) collaboration at Baylor College of Medicine, the vaccine was studied in rhesus macaques and, in at least some animals, seemed to generate multivalent T cell responses that apparently correlated with better control of viral load after challenge with a hybrid simian-human immunodeficiency virus (SHIV).
Prior vaccination increases the epitopic breadth of the cytotoxic T-lymphocyte response that evolves in rhesus monkeys following a simian-human immunodeficiency virus infection.